Sestrin regulation of TORC1: Is Sestrin a leucine sensor?-Reference-Cited by-同舟云学术

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Sestrin regulation of TORC1: Is Sestrin a leucine sensor?

Published:2016-06-07 Issue:431 Volume:9 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Lee Jun Hee¹,Cho Uhn-Soo²,Karin Michael³

Affiliation:

1. Department of Molecular & Integrative Physiology, University of Michigan, Ann Arbor, MI 48109, USA.

2. Department of Biological Chemistry, University of Michigan, Ann Arbor, MI 48109, USA.

3. Departments of Pharmacology and Pathology, University of California, San Diego, School of Medicine, La Jolla, CA 92093–0723, USA.

Abstract

Sestrin2 may not be a leucine sensor for mTORC1.

Funder

NIH

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference44 articles.

1. PA26, a novel target of the p53 tumor suppressor and member of the GADD family of DNA damage and growth arrest inducible genes

2. Identification of a novel stress-responsive gene Hi95 involved in regulation of cell viability

3. Sestrins Orchestrate Cellular Metabolism to Attenuate Aging

4. p53 Target Genes Sestrin1 and Sestrin2 Connect Genotoxic Stress and mTOR Signaling

5. Sestrin2 Modulates AMPK Subunit Expression and Its Response to Ionizing Radiation in Breast Cancer Cells

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