A Mouse Model of Familial Hypertrophic Cardiomyopathy

Author:

Geisterfer-Lowrance Anja A. T.1,Christe Michael1,Conner David A.1,Ingwall Joanne S.2,Schoen Frederick J.3,Seidman Christine E.4,Seidman J. G.1

Affiliation:

1. A. A. T. Geisterfer-Lowrance, M. Christe, D. A. Conner, J. G. Seidman, Howard Hughes Medical Institute and Department of Genetics, Harvard Medical School, 200 Longwood Avenue, Boston, MA 02115, USA.

2. J. S. Ingwall, Department of Medicine, Brigham and Women's Hospital, 75 Francis Street, Boston, MA 02115, USA.

3. F. J. Schoen, Department of Pathology, Brigham and Women's Hospital, 75 Francis Street, Boston, MA 02115, USA.

4. C. E. Seidman, Howard Hughes Medical Institute, Brigham and Women's Hospital, 75 Francis Street, Boston MA 02115, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference18 articles.

1. CHRISTE M unpublished data.

2. CILAZAPRIL TREATMENT DEPRESSES VENTRICULAR-FUNCTION IN SPONTANEOUSLY HYPERTENSIVE RATS;CHRISTE M.E.;AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY,1994

3. GEISTERFERLOWRA AA unpublished data.

4. INTRODUCTION OF A SUBTLE MUTATION INTO THE HOX-2.6 LOCUS IN EMBRYONIC STEM-CELLS;HASTY P;NATURE,1991

5. ALTERED HEPATIC TRANSPORT OF IMMUNOGLOBULIN-A IN MICE LACKING THE J-CHAIN;HENDRICKSON B.A.;JOURNAL OF EXPERIMENTAL MEDICINE,1995

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