Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege

Author:

Griffith Thomas S.1,Brunner Thomas2,Fletcher Sharon M.1,Green Douglas R.2,Ferguson Thomas A.3

Affiliation:

1. T. S. Griffith and S. M. Fletcher, Department of Ophthalmology and Visual Sciences, Washington University School of Medicine, St. Louis, MO 63110, USA.

2. T. Brunner and D. R. Green, Division of Cellular Immunology, La Jolla Institute for Allergy and Immunology, La Jolla, CA 92037, USA.

3. T. A. Ferguson, Departments of Ophthalmology and Visual Sciences and Pathology, Washington University School of Medicine, St. Louis, MO 63110, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference26 articles.

1. RETINITIS AND DEVIANT IMMUNE-RESPONSES FOLLOWING INTRAVITREAL INOCULATION OF HSV-1;ATHERTON S.S.;INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE,1987

2. CELL-AUTONOMOUS FAS (CD95) FAS-LIGAND INTERACTION MEDIATES ACTIVATION-INDUCED APOPTOSIS IN T-CELL HYBRIDOMAS;BRUNNER T;NATURE,1995

3. T-CELL LINES MEDIATING EXPERIMENTAL AUTOIMMUNE UVEORETINITIS (EAU) IN THE RAT;CASPI R.R.;JOURNAL OF IMMUNOLOGY,1986

4. NEUROPEPTIDES MODULATE IMMUNE DEVIATION INDUCED VIA THE ANTERIOR-CHAMBER OF THE EYE;FERGUSON T.A.;JOURNAL OF IMMUNOLOGY,1995

5. GAO E.K. unpublished data.

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