Chronic Mucocutaneous Candidiasis in Humans with Inborn Errors of Interleukin-17 Immunity-Reference-Cited by-同舟云学术

Chronic Mucocutaneous Candidiasis in Humans with Inborn Errors of Interleukin-17 Immunity

Published:2011-04 Issue:6025 Volume:332 Page:65-68
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Puel Anne¹,Cypowyj Sophie²,Bustamante Jacinta¹,Wright Jill F.³,Liu Luyan¹,Lim Hye Kyung²,Migaud Mélanie¹,Israel Laura¹,Chrabieh Maya¹,Audry Magali²,Gumbleton Matthew⁴,Toulon Antoine⁵,Bodemer Christine⁵,El-Baghdadi Jamila⁶,Whitters Matthew³,Paradis Theresa³,Brooks Jonathan³,Collins Mary³,Wolfman Neil M.³,Al-Muhsen Saleh⁷,Galicchio Miguel⁸,Abel Laurent¹²,Picard Capucine¹⁹¹⁰,Casanova Jean-Laurent¹²⁷¹⁰

Affiliation:

1. Laboratory of Human Genetics of Infectious Diseases, Necker Branch, Institut National de la Santé et de la Recherche Médicale, U980, and University Paris Descartes, Necker Medical School, 75015 Paris, France.

2. St. Giles Laboratory of Human Genetics of Infectious Diseases, Rockefeller Branch, The Rockefeller University, New York, NY 10065, USA.

3. Inflammation and Immunology, Pfizer Research, Cambridge, MA 02140, USA.

4. SUNY Upstate Medical University, Syracuse, NY 13210, USA.

5. Dermatology Unit, Necker Hospital, 75015 Paris, France.

6. Unit of Genetics, Military Hospital of Instruction Mohamed V, Rabat 10000, Morocco.

7. Prince Naif Center for Immunology Research, Department of Pediatrics, College of Medicine, King Saud University, Riyadh 11461, Saudi Arabia.

8. Victor J. Vilela Children’s Hospital, Rosario, Santa Fe 2000, Argentina.

9. Center for the Study of Primary Immunodeficiencies, Necker Hospital, 75015 Paris, France.

10. Pediatric Hematology-Immunology Unit, Necker Hospital, Paris 75015, France.

Abstract

Chronic yeast infections in the absence of other infections result from genetic deficiencies in proinflammatory host responses.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference30 articles.

1. Inborn errors of mucocutaneous immunity to Candida albicans in humans: a role for IL-17 cytokines?

2. Mutations in STAT3 and IL12RB1 impair the development of human IL-17–producing T cells

3. Deficiency of Th17 cells in hyper IgE syndrome due to mutations in STAT3

4. Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndrome

5. Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

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