Synaptic Plasticity in Spinal Lamina I Projection Neurons That Mediate Hyperalgesia
Author:
Affiliation:
1. Institute of Physiology and Pathophysiology, Heidelberg University, D-69120 Heidelberg, Germany.
2. Brain Research Institute, Vienna University Medical School, A-1090 Vienna, Austria.
Abstract
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference27 articles.
1. Transmission of Chronic Nociception by Spinal Neurons Expressing the Substance P Receptor
2. Inhibition of Hyperalgesia by Ablation of Lamina I Spinal Neurons Expressing the Substance P Receptor
3. Spinal Substance P Receptor Expression and Internalization in Acute, Short-Term, and Long-Term Inflammatory Pain States
4. Cutaneous stimuli releasing immunoreactive substance P in the dorsal horn of the cat
5. Spinal Neurons that Possess the Substance P Receptor Are Required for the Development of Central Sensitization
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