Abnormal Development of Peripheral Lymphoid Organs in Mice Deficient in Lymphotoxin

Author:

De Togni Pietro1,Goellner Joseph2,Ruddle Nancy H.3,Streeter Philip R.2,Fick Andrea4,Mariathasan Sanjeev4,Smith Stacy C.1,Carlson Rebecca4,Shornick Laurie P.1,Strauss-Schoenberger Jena2,Russell John H.5,Karr Robert2,Chaplin David D.4

Affiliation:

1. Department of Internal Medicine, Washington University School of Medicine, St. Louis, MO 63110, USA.

2. Department of Immunology, Monsanto Company, St. Louis, MO 63198, USA.

3. Department of Epidemiology and Public Health, Yale University School of Medicine, New Haven, CT 06510, USA.

4. Howard Hughes Medical Institute, Washington University School of Medicine, St. Louis, MO 63110, USA.

5. Department of Molecular Biology and Pharmacology, Washington University School of Medicine, St. Louis, MO 63110, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference36 articles.

1. BROUDY, V.C., DISPARATE EFFECTS OF TUMOR-NECROSIS-FACTOR-ALPHA CACHECTIN AND TUMOR NECROSIS FACTOR-BETA LYMPHOTOXIN ON HEMATOPOIETIC GROWTH-FACTOR PRODUCTION AND NEUTROPHIL ADHESION MOLECULE EXPRESSION BY CULTURED HUMAN-ENDOTHELIAL CELLS, JOURNAL OF IMMUNOLOGY 138: 4298 (1987).

2. BROWNINGJL, CELL 72: 847 (1993).

3. CASEY, M.L., CACHECTIN TUMOR NECROSIS FACTOR-ALPHA FORMATION IN HUMAN DECIDUA - POTENTIAL ROLE OF CYTOKINES IN INFECTION-INDUCED PRETERM LABOR, JOURNAL OF CLINICAL INVESTIGATION 83: 430 (1989).

4. Chaplin, D. D., Proceedings of the National Academy of Sciences of the United States of America 80: 6947 (1983).

5. CONTA, B.S., ACTIVATION OF LYT-1+ AND LYT-2+ T-CELL CLONED LINES - STIMULATION OF PROLIFERATION, LYMPHOKINE PRODUCTION, AND SELF-DESTRUCTION, JOURNAL OF IMMUNOLOGY 134: 2185 (1985).

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