Putative PPAR Target Genes Express Highly in Skeletal Muscle of Insulin-Resistant MetS Model SHR/NDmc-cp Rats

Author:

HARIYA Natsuyo12,MIYAKE Kunio1,KUBOTA Takeo1,GODA Toshinao3,MOCHIZUKI Kazuki43

Affiliation:

1. Department of Epigenetic Medicine, Interdisciplinary Graduate School of Medicine and Engineering, University of Yamanashi

2. Interdisciplinary Graduate School of Medicine and Engineering, University of Yamanashi

3. School of Food and Nutritional Sciences, University of Shizuoka

4. Laboratory of Food and Nutritional Sciences, Department of Local Produce and Food Sciences, Faculty of Life and Environmental Sciences, University of Yamanashi

Publisher

Center for Academic Publications Japan

Subject

Nutrition and Dietetics,Medicine (miscellaneous)

Reference29 articles.

1. 1) DeFronzo RA. 1988. Lilly lecture 1987. The triumvirate: β-Cell, muscle, liver. A collusion responsible for NIDDM. Diabetes 37: 667-687.

2. 2) Gorter PM, Olijhoek JK, van der Graaf Y, Algra A, Rabelink TJ, Visseren FL. 2004. Prevalence of the metabolic syndrome in patients with coronary heart disease, cerebrovascular disease, peripheral arterial disease or abdominal aortic aneurysm. Atherosclerosis 173: 363-369.

3. 3) Watson RT, Pessin JE. 2001. Intracellular organization of insulin signaling and GLUT4 translocation. Recent Prog Horm Res 56: 175-193.

4. 4) Long YC, Zierath JR. 2006. AMP-activated protein kinase signaling in metabolic regulation. J Clin Invest 116: 1776-1783.

5. 5) Alibegovic AC, Sonne MP, Højbjerre L, Bork-Jensen J, Jacobsen S, Nilsson E, Faerch K, Hiscock N, Mortensen B, Friedrichsen M, Stallnecht B, Dela F, Vaag A. 2010. Insulin resistance induced by physical inactivity is associated with multiple transcriptional changes in skeletal muscle in young men. Am J Physiol Endocrinol Metab 299: E752-763.

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