Characterization of Usher Syndrome Cell line Genotypes and Elucidation of Novel MicroRNA Biomarkers using MicroRNA Microarray and Droplet Digital PCR

Author:

Tom Wesley1,Chandel Dinesh S.1,Jiang Chao1,Krzyzanowski Gary1,Fernando Nirmalee1,Olou Appolinaire1,Fernando M. Rohan1

Affiliation:

1. Boys Town National Research Hospital

Abstract

Abstract

Background:Usher syndrome (USH) is an inherited disorder characterized by sensorineural hearing loss (SNHL), retinitis pigmentosa (RP)-related vision loss, and vestibular dysfunction. USH presents itself as three distinct clinical types 1, 2 and 3, with no biomarker for early detection. This study aimed to explore novel microRNA (miRNA) biomarkers for USH by comparing miRNA expression patterns in cell lines derived from USH patients and control subjects. Methods:Lymphocytes from USH patients and healthy individuals were isolated and transformed into stable cell lines using Epstein-Barr virus (EBV). DNA from these cell lines was sequenced using a targeted panel to identify gene variants associated with USH types 1, 2, and 3. Microarray analysis was performed on RNA from both USH and control cell lines using NanoString miRNA microarray technology. Dysregulated miRNAs identified by the microarray were validated using droplet digital PCR technology. Results: DNA sequencing revealed that two USH patients had USH type 1 with gene variants in USH1B (MYO7A) and USH1D (CDH23), while the other two patients were classified as USH type 2 (USH2A) and USH type 3 (CLRN-1), respectively. The NanoString miRNA microarray detected 92 differentially expressed miRNAs in USH cell lines compared to controls. Significantly altered miRNAs exhibited at least a twofold increase or decrease with a p value below 0.05. Among these miRNAs, 20 were specific to USH1, 14 to USH2, and 5 to USH3. Three miRNAs that are known as miRNA-183-family which are crucial for inner ear and retina development have been significantly down regulated as compared to control cells. Subsequently, droplet digital PCR assays confirmed the dysregulation of twelve most prominent miRNAs in USH cell lines. Conclusion:This study identifies several miRNAs with differential expression in USH patients and their potential utility as biomarkers for Usher syndrome.

Publisher

Springer Science and Business Media LLC

Reference77 articles.

1. Usher Syndrome: Genetics and Molecular Links of Hearing Loss and Directions for Therapy;Whatley M;Front. Genet.,2020

2. The genetic and phenotypic landscapes of Usher syndrome: from disease mechanisms to a new classification;Delmaghani S;Hum. Genet.,2022

3. Frequency of Usher syndrome in two pediatric populations: Implications for genetic screening of deaf and hard of hearing children;Kimberling WJ;Genet. Med. Off. J. Am. Coll. Med. Genet.,2010

4. Digenic inheritance of deafness caused by mutations in genes encoding cadherin 23 and protocadherin 15 in mice and humans;Zheng QY;Hum. Mol. Genet.,2005

5. Digenic inheritance of deafness caused by 8J allele of myosin-VIIA and mutations in other Usher I genes;Zheng QY;Hum. Mol. Genet.,2012

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号  京ICP备18003416号-3