Wnts synergize to activate β-catenin signaling
Author:
Affiliation:
1. Programme in Cancer and Stem Cell Biology, Duke-NUS Medical School, Singapore 169857
2. Centre for Computational Biology, Duke-NUS Medical School, Singapore 169857
3. Department of Pediatrics, Duke University School of Medicine, Durham, NC, USA
Abstract
Funder
National Medical Research Council
National Research Foundation Singapore
Publisher
The Company of Biologists
Subject
Cell Biology
Link
http://journals.biologists.com/jcs/article-pdf/130/9/1532/1946544/jcs198093.pdf
Reference55 articles.
1. Wnt7b is required for epithelial progenitor growth and operates during epithelial-to-mesenchymal signaling in pancreatic development;Afelik;Dev. Biol.,2015
2. Wnt pathway aberrations including autocrine Wnt activation occur at high frequency in human non-small-cell lung carcinoma;Akiri;Oncogene,2009
3. WNT signalling pathways as therapeutic targets in cancer;Anastas;Nat. Rev. Cancer,2013
4. Angiogenic sprouting into neural tissue requires Gpr124, an orphan G protein-coupled receptor;Anderson;Proc. Natl Acad. Sci. USA,2011
5. WNT7B mediates autocrine Wnt/β-catenin signaling and anchorage-independent growth in pancreatic adenocarcinoma;Arensman;Oncogene,2014
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