Polarity proteins PAR6 and aPKC regulate cell death through GSK-3β in 3D epithelial morphogenesis
Author:
Affiliation:
1. Departments of Anatomy, and Biochemistry and Biophysics, University of California School of Medicine, San Francisco, CA 94158, USA
2. Department of Pediatrics, University of California School of Medicine, San Francisco, CA, USA
Abstract
Publisher
The Company of Biologists
Subject
Cell Biology
Link
http://journals.biologists.com/jcs/article-pdf/120/14/2309/1510718/2309.pdf
Reference59 articles.
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2. Aranda, V., Haire, T., Nolan, M. E., Calarco, J. P., Rosenberg, A. Z., Fawcett, J. P., Pawson, T. and Muthuswamy, S. K. (2006). Par6-aPKC uncouples ErbB2 induced disruption of polarized epithelial organization from proliferation control. Nat. Cell Biol.8, 1235-1245.
3. Bandyopadhyay, G., Standaert, M. L., Sajan, M. P., Kanoh, Y., Miura, A., Braun, U., Kruse, F., Leitges, M. and Farese, R. V. (2004). Protein kinase C-lambda knockout in embryonic stem cells and adipocytes impairs insulin-stimulated glucose transport. Mol. Endocrinol.18, 373-383.
4. Betschinger, J., Mechtler, K. and Knoblich, J. A. (2003). The Par complex directs asymmetric cell division by phosphorylating the cytoskeletal protein Lgl. Nature422, 326-330.
5. Bilder, D. (2004). Epithelial polarity and proliferation control: links from the Drosophila neoplastic tumor suppressors. Genes Dev.18, 1909-1925.
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