Use of a Physiologically Based Pharmacokinetic Model for Rats to Study the Influence of Body Fat Mass and Induction of CYP1A2 on the Pharmacokinetics of TCDD-Reference-Cited by-同舟云学术

Use of a Physiologically Based Pharmacokinetic Model for Rats to Study the Influence of Body Fat Mass and Induction of CYP1A2 on the Pharmacokinetics of TCDD

Published:2006-09 Issue:9 Volume:114 Page:1394-1400
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environ Health Perspect

Author:

Emond Claude¹²³,Birnbaum Linda S.²,DeVito Michael J.²

Affiliation:

1. National Research Council, National Academy of Sciences, Washington, DC, USA

2. National Health and Environmental Effects Research Laboratory, U.S. Environmental Protection Agency, Research Triangle Park, North Carolina, USA

3. Environmental and Occupational Health Department, Medicine Faculty, University of Montreal, Montreal, Quebec, Canada

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference47 articles.

1. Severe 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) intoxication: Insights into the measurement of hepatic cytochrome P450 1A2 induction*

2. Pharmacokinetics and biological activity of 2,3,7,8-tetrachlorodibenzo-p-dioxin

3. Tissue distribution, excretion and biological effects of in rats

4. Regional Hepatic CYP1A1 and CYP1A2 Induction with 2,3,7,8-Tetrachlorodibenzo-p-dioxin Evaluated with a Multicompartment Geometric Model of Hepatic Zonation

5. Modeling Receptor-Mediated Processes with Dioxin: Implications for Pharmacokinetics and Risk Assessment

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