Differential Response to Trichloroethylene-Induced Hepatosteatosis in Wild-Type and PPARα-Humanized Mice-Reference-Cited by-同舟云学术

Differential Response to Trichloroethylene-Induced Hepatosteatosis in Wild-Type and PPARα-Humanized Mice

Published:2010-11 Issue:11 Volume:118 Page:1557-1563
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environ Health Perspect

Author:

Ramdhan Doni Hikmat¹,Kamijima Michihiro¹²,Wang Dong¹,Ito Yuki¹²,Naito Hisao¹,Yanagiba Yukie¹,Hayashi Yumi¹,Tanaka Naoki³,Aoyama Toshifumi³,Gonzalez Frank J.⁴,Nakajima Tamie¹

Affiliation:

1. Department of Occupational and Environmental Health, Nagoya University Graduate School of Medicine, Nagoya, Japan

2. Department of Occupational and Environmental Health, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan

3. Department of Metabolic Regulation, Shinshu University Graduate School of Medicine, Matsumoto, Japan

4. Laboratory of Metabolism, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference42 articles.

1. Altered Constitutive Expression of Fatty Acid-metabolizing Enzymes in Mice Lacking the Peroxisome Proliferator-activated Receptor α (PPARα)

2. The Mechanisms of Action of PPARs

3. Nonalcoholic Steatohepatitis: A Proposal for Grading and Staging The Histological Lesions

4. Diminished Hepatocellular Proliferation in Mice Humanized for the Nuclear Receptor Peroxisome Proliferator-Activated Receptor α

5. Peroxisome Proliferator-Activated Receptors (PPAR) and the Mitochondrial Aldehyde Dehydrogenase (ALDH2) Promoter In Vitro and In Vivo

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