DMXAA Causes Tumor Site-Specific Vascular Disruption in Murine Non-Small Cell Lung Cancer, and like the Endogenous Non-Canonical Cyclic Dinucleotide STING Agonist, 2′3′-cGAMP, Induces M2 Macrophage Repolarization-Reference-Cited by-同舟云学术

DMXAA Causes Tumor Site-Specific Vascular Disruption in Murine Non-Small Cell Lung Cancer, and like the Endogenous Non-Canonical Cyclic Dinucleotide STING Agonist, 2′3′-cGAMP, Induces M2 Macrophage Repolarization

Published:2014-06-18 Issue:6 Volume:9 Page:e99988
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Downey Charlene M.,Aghaei Mehrnoosh,Schwendener Reto A.,Jirik Frank R.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

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5. The antitumour activity of 5,6-dimethylxanthenone-4-acetic acid (DMXAA) in TNF receptor-1 knockout mice;L Zhao;British Journal of Cancer,2002

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