Clostridium perfringens Beta-Toxin Induces Necrostatin-Inhibitable, Calpain-Dependent Necrosis in Primary Porcine Endothelial Cells
Author:
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference37 articles.
1. Beta toxin is essential for the intestinal virulence of Clostridium perfringens type C disease isolate CN3685 in a rabbit ileal loop model;S Sayeed;Mol Microbiol,2008
2. Recent progress in understanding the pathogenesis of Clostridium perfringens type C infections;FA Uzal;Vet Microbiol,2011
3. Molecular genetic analysis of beta-toxin of Clostridium perfringens reveals sequence homology with alpha-toxin, gamma-toxin, and leukocidin of Staphylococcus aureus;SE Hunter;Infect Immun,1993
4. Biological activities and pore formation of Clostridium perfringens beta toxin in HL 60 cells;M Nagahama;J Biol Chem,2003
5. Clostridium perfringens beta-toxin forms potential-dependent, cation-selective channels in lipid bilayers;O Shatursky;Infect Immun,2000
Cited by 48 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Clostridial diarrheas in piglets: A review;Veterinary Microbiology;2023-05
2. Decreased S100A9 expression alleviates Clostridium perfringens beta2 toxin-induced inflammatory injury in IPEC-J2 cells;PeerJ;2023-01-25
3. The acidity of oxalic acid induces regulated cell death in Arabidopsis;2022-11-03
4. LncRNA ALDB-898 modulates intestinal epithelial cell damage caused by Clostridium perfringens type C in piglet by regulating ssc-miR-122–5p/OCLN signaling;Molecular Immunology;2022-09
5. Regulated necrosis, a proinflammatory cell death, potentially counteracts pathogenic infections;Cell Death & Disease;2022-07-22
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3