Phosphoinositide 3-kinase/Akt pathway is involved in pingyangmycin-induced growth inhibition, apoptosis and reduction of invasive potential in EOMA mouse hemangioendothelioma cells
Author:
Publisher
Spandidos Publications
Subject
Cancer Research,Oncology,Genetics,Molecular Biology,Molecular Medicine,Biochemistry
Cited by 11 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. MicroRNA‑203a‑3p improves bleomycin and pingyangmycin sensitivity by inactivating the PI3K/AKT pathway in hemangioma;Experimental and Therapeutic Medicine;2024-01-04
2. Trop-2 and Nectin-4 immunohistochemical expression in metastatic colorectal cancer: searching for the right population for drugs’ development;British Journal of Cancer;2023-02-09
3. Pingyangmycin Activates Oral Carcinoma Cell Autophagy via the Phosphorylation of the PI3K/AKT/mTOR Axis to Achieve the Purpose of Treating Oral Carcinoma;Emergency Medicine International;2022-08-28
4. Oral Delivery of Pingyangmycin by Layer-by-Layer (LbL) Self-Assembly Polyelectrolyte-Grafted Nano Graphene Oxide;Journal of Nanoscience and Nanotechnology;2019-04-01
5. Knockdown of inhibitor of differentiation 1 suppresses proliferation and induces apoptosis by inactivating PI3K/Akt/mTOR signaling in hemangioma-derived endothelial cells;Biomedicine & Pharmacotherapy;2019-03
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