JMJD3 deficiency alleviates lipopolysaccharide‑induced acute lung injury by inhibiting alveolar epithelial ferroptosis in a Nrf2‑dependent manner
Author:
Affiliation:
1. Department of Cardiothoracic Surgery, Suizhou Hospital, Hubei University of Medicine, Suizhou, Hubei 441300, P.R. China
Publisher
Spandidos Publications
Subject
Cancer Research,Oncology,Genetics,Molecular Biology,Molecular Medicine,Biochemistry
Cited by 10 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Knockdown of JMJD3 ameliorates cigarette smoke extract-triggered bronchial epithelial cell injury via ACSL4-dependent ferroptosis;Toxicology in Vitro;2024-02
2. TMEM16A deficiency in alveolar type 2 epithelial cells protected against endoplasmic reticulum stress-induced ferroptosis during acute lung injury;International Immunopharmacology;2023-12
3. Ficolin B secreted by alveolar macrophage exosomes exacerbates bleomycin-induced lung injury via ferroptosis through the cGAS-STING signaling pathway;Cell Death & Disease;2023-08-30
4. Knockdown of SHP2 attenuated LPS-induced ferroptosis via downregulating ACSL4 expression in acute lung injury;Allergologia et Immunopathologia;2023-05-01
5. Tripartite Motif Containing 37 (TRIM37 )-Mediated Ubiquitination of Forkhead Box Transcription Factor O1 (FOXO1) Elevates Lipopolysaccharide-Induced Ferroptosis in Lung Endothelial Cells;J BIOL REG HOMEOS AG;2023
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