Loganin Prevents Hepatic Steatosis by Blocking NLRP3 Inflammasome Activation
Author:
Affiliation:
1. College of Pharmacy, The Catholic University of Korea, Bucheon 14662, Republic of Korea
2. Department of Pharmacology, College of Korean Medicine, Woosuk University, Jeonju 54986, Republic of Korea
Funder
National Research Foundation of Korea
Ministry of Science, ICT and Future Planning
Publisher
The Korean Society of Applied Pharmacology
Subject
Drug Discovery,Pharmacology,Molecular Medicine,Biochemistry
Link
http://pdf.medrang.co.kr/BT2/2023/031/BT031-01-40.pdf
Reference29 articles.
1. The role of nutrition in non‐alcoholic fatty liver disease: Pathophysiology and management
2. Differential splicing of the apoptosis-associated speck like protein containing a caspase recruitment domain (ASC) regulates inflammasomes
3. Activation of Inflammasomes Requires Intracellular Redistribution of the Apoptotic Speck-Like Protein Containing a Caspase Recruitment Domain
4. Loganin Attenuates High Glucose-Induced Schwann Cells Pyroptosis by Inhibiting ROS Generation and NLRP3 Inflammasome Activation
5. Non-alcoholic fatty liver disease associated with hepatocellular carcinoma: An increasing concern
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1. Loganin alleviates myocardial ischemia–reperfusion injury through GLP‐1R/NLRP3‐mediated pyroptosis pathway;Environmental Toxicology;2023-07-27
2. NOD-like receptors in the pathogenesis of metabolic (dysfunction)-associated fatty liver disease: Therapeutic agents targeting NOD-like receptors;Diabetes & Metabolic Syndrome: Clinical Research & Reviews;2023-07
3. A potential therapeutic approach for ulcerative colitis: targeted regulation of macrophage polarization through phytochemicals;Frontiers in Immunology;2023-05-01
4. Inhibitory Effects of Loganin on Adipogenesis In Vitro and In Vivo;International Journal of Molecular Sciences;2023-03-01
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