Qiliqiangxin Attenuates Oxidative Stress-Induced Mitochondrion-Dependent Apoptosis in Cardiomyocytes <i>via</i> PI3K/AKT/GSK3β Signaling Pathway-Reference-Cited by-同舟云学术

Qiliqiangxin Attenuates Oxidative Stress-Induced Mitochondrion-Dependent Apoptosis in Cardiomyocytes via PI3K/AKT/GSK3β Signaling Pathway

Published:2019-08-01 Issue:8 Volume:42 Page:1310-1321
ISSN:0918-6158
Container-title:Biological and Pharmaceutical Bulletin
language:en
Short-container-title:Biol. Pharm. Bull.

Author:

Zhao Qifei¹,Li Hongrong¹,Chang Liping²³,Wei Cong³⁴,Yin Yujie²,Bei Hongying²,Wang Zhixin³⁴,Liang Junqing³⁵,Wu Yiling¹²

Affiliation:

1. Department of Integrated Traditional Chinese and Western Medicine, Hebei Medical University

2. Key Disciplines of State Administration of TCM for Collateral Disease, Affiliated Yiling Hospital of Hebei Medical University

3. National Key Laboratory of Collateral Disease Research and Innovative Chinese Medicine, Hebei Yiling Pharmaceutical Research Institute

4. Key Laboratory of State Administration of TCM (Cardio-Cerebral Vessel Collateral Disease)

5. Key Laboratory of Hebei Province for Collateral Diseases

Publisher

Pharmaceutical Society of Japan

Subject

Pharmaceutical Science,Pharmacology,General Medicine

Link

https://www.jstage.jst.go.jp/article/bpb/42/8/42_b19-00050/_pdf

Reference41 articles.

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3. 3) Hou L, Guo J, Xu F, Weng X, Yue W, Ge J. Cardiomyocyte dimethylarginine dimethylaminohydrolase1 attenuates left-ventricular remodeling after acute myocardial infarction: involvement in oxidative stress and apoptosis. Basic Res. Cardiol., 113, 28 (2018).

4. 4) van der Pol A, van Gilst WH, Voors AA, van der Meer P. Treating oxidative stress in heart failure: past, present and future. Eur. J. Heart Fail., 21, 425–435 (2019).

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