High Affinity Molecules Disrupting GRB2 Protein Complexes as a Therapeutic Strategy for Chronic Myelogenous Leukaemia-Reference-Cited by-同舟云学术

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High Affinity Molecules Disrupting GRB2 Protein Complexes as a Therapeutic Strategy for Chronic Myelogenous Leukaemia

Published:2003-01 Issue:3 Volume:44 Page:411-427
ISSN:1042-8194
Container-title:Leukemia & Lymphoma
language:en
Short-container-title:Leukemia & Lymphoma

Author:

Feller Stephan M.,Tuchscherer Gabriele,Voss Jan

Publisher

Informa UK Limited

Subject

Cancer Research,Oncology,Hematology

Link

http://www.tandfonline.com/doi/pdf/10.1080/1042819021000037930

Reference262 articles.

1. The Biology of Chronic Myeloid Leukemia

2. Chronic Myeloid Leukemia

3. Molecular pathogenesis of chronic myeloid leukemia: implications for new therapeutic strategies

4. Leukemic kinases of the Abl family− an update

5. The t(8;22) in chronic myeloid leukemia fuses BCR to FGFR1: transforming activity and specific inhibition of FGFR1 fusion proteins

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1. GRB2: A dynamic adaptor protein orchestrating cellular signaling in health and disease;Biochemistry and Biophysics Reports;2024-09

2. Potential pharmacological mechanisms of tanshinone IIA in the treatment of human neuroblastoma based on network pharmacological and molecular docking Technology;Frontiers in Pharmacology;2024-03-11

3. The Role of Grb2 in Cancer and Peptides as Grb2 Antagonists;Protein & Peptide Letters;2018-02-08

4. Insights into the Shc Family of Adaptor Proteins;Journal of Molecular Signaling;2017-05-03

5. SH3 Domains as Drug Targets;Protein-Protein Interactions in Drug Discovery;2013-02-25

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