HPV16 E6-Induced and E6AP-Dependent Inhibition of the Transcriptional Coactivator hADA3 in Human Cervical Carcinoma Cells
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1080/07357900802350798
Reference22 articles.
1. Papillomaviruses Causing Cancer: Evasion From Host-Cell Control in Early Events in Carcinogenesis
2. The Human Papillomavirus E6 protein and its contribution to malignant progression
3. Human papillomavirus-induced carcinogenesis and the ubiquitin–proteasome system
4. The Role of E6AP in the Regulation of p53 Protein Levels in Human Papillomavirus (HPV)-positive and HPV-negative Cells
5. The Role of the Ubiquitin Ligase E6-AP in Human Papillomavirus E6-mediated Degradation of PDZ Domain-containing Proteins
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1. Mechanistic insights into the oncogenic partnership of hADA3 and HPVE6 - paving ways for improved cervical cancer therapy;Indian Journal of Biochemistry and Biophysics;2023
2. Anti‑Condyloma acuminata mechanism of microRNAs‑375 modulates HPV in cervical cancer cells via the UBE3A and IGF‑1R pathway;Oncology Letters;2018-06-18
3. In silico modeling of the cryptic E2∼ubiquitin–binding site of E6-associated protein (E6AP)/UBE3A reveals the mechanism of polyubiquitin chain assembly;Journal of Biological Chemistry;2017-11
4. Molecular Probing of the HPV-16 E6 Protein Alpha Helix Binding Groove with Small Molecule Inhibitors;PLOS ONE;2016-02-25
5. High-risk HPV16E6 stimulates hADA3 degradation by enhancing its SUMOylation;Carcinogenesis;2014-05-02
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