Podocytes maintain high basal levels of autophagy independent of mtor signaling

Author:

Bork Tillmann1,Liang Wei12,Yamahara Kosuke13,Lee Philipp1,Tian Zhejia1,Liu Shuya4,Schell Christoph156,Thedieck Kathrin789,Hartleben Bjoern10,Patel Ketan1112,Tharaux Pierre-Louis1314ORCID,Lenoir Olivia13,Huber Tobias B.4

Affiliation:

1. Department of Medicine IV, Faculty of Medicine, University of Freiburg, Freiburg, Germany

2. Division of Nephrology, Renmin Hospital of Wuhan University, Wuhan, China

3. Department of Medicine, Shiga University of Medical Science, Otsu, Japan

4. III. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany

5. Institute of Surgical Pathology, Faculty of Medicine, University of Freiburg, Freiburg, Germany

6. Berta-Ottenstein Programme, Faculty of Medicine, University of Freiburg, Freiburg, Germany

7. Institute of Biochemistry and Center for Molecular Biosciences Innsbruck, University of Innsbruck, Innsbruck, Austria

8. Department of Pediatrics, Section Systems Medicine of Metabolism and Signaling, University of Groningen, University Medical Center Groningen (UMCG), Groningen, The Netherlands

9. Department of Neuroscience, School of Medicine and Health Sciences, Carl von Ossietzky University Oldenburg, Oldenburg, Germany

10. Institute of Pathology, Hannover Medical School, Hannover, Germany

11. School of Biological Science, University of Reading, Reading, UK

12. FFRIAS, Freiburg Institute for Advanced Studies, Albert-Ludwigs-University, Freiburg, Germany

13. PARCC, INSERM, Université de Paris, Paris, France

14. Nephrology Division, Georges Pompidou European Hospital, Paris, France

Funder

German Research Foundation Heisenberg Program

H2020-IMI2 BEAt-DKD

BMBF-STOP-FSGS

German Research Foundation

National Natural Science Foundation of China

BMBF GlioPATH

BMBF MAPTor-NET

MESI-STRAT

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

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