Extracellular stimulation of VSIG4/complement receptor Ig suppresses intracellular bacterial infection by inducing autophagy-Reference-Cited by-同舟云学术

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Extracellular stimulation of VSIG4/complement receptor Ig suppresses intracellular bacterial infection by inducing autophagy

Published:2016-08-11 Issue:9 Volume:12 Page:1647-1659
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Kim Kwang H.¹,Choi Beom K.²,Kim Young H.³,Han Chungyong²,Oh Ho S.²,Lee Don G.²,Kwon Byoung S.¹²⁴

Affiliation:

1. Eutilex, The Catholic University School of Medicine Seoul, Korea

2. Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Goyang, Korea

3. Immune Cell Production Unit, Program for Immunotherapeutic Research, National Cancer Center, Goyang, Korea

4. Department of Medicine, Tulane University Health Sciences Center, New Orleans, LA, USA

Funder

National Research Foundation of Korea (Byoung S. Kwon, bskwon@ncc.re.kr)

National Cancer Korea (Byoung S. Kwon, bskwon@ncc.re.kr)

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2016.1196314

Reference48 articles.

1. The interface between innate and adaptive immunity

2. Interleukin 1 is released by murine macrophages during apoptosis induced by Shigella flexneri.

3. Two inflammatory mediator cytokine genes are closely linked and variably amplified on chromosome 17q

4. Antigen-Presenting Function of the Macrophage

5. Distinct roles of class I and class III phosphatidylinositol 3-kinases in phagosome formation and maturation

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