CAMK2/CaMKII activates MLKL in short-term starvation to facilitate autophagic flux

Author:

Zhan Qionghui123,Jeon Jaepyo3,Li Ying4,Huang Yu35,Xiong Jian35ORCID,Wang Qiaochu3ORCID,Xu Tian-Le67,Li Yong2,Ji Fu-Hai1,Du Guangwei35ORCID,Zhu Michael X35ORCID

Affiliation:

1. Department of Anesthesiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China

2. Department of Biochemistry and Molecular Cell Biology, Shanghai Key Laboratory for Tumor Microenvironment and Inflammation, Institute of Medical Sciences, Shanghai Jiao Tong University School of Medicine, Shanghai, China

3. Department of Integrative Biology and Pharmacology, McGovern Medical School, the University of Texas Health Science Center at Houston, Houston, Texas, USA

4. Department of Anesthesia, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

5. Program in Biochemistry and Cell Biology, MD Anderson Cancer Center and UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA

6. Center for Brain Science, Shanghai Children’s Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, China

7. Department of Anatomy and Physiology, Shanghai Jiao Tong University School of Medicine, Shanghai, China

Funder

American Heart Association

National Natural Science Foundation of China

Natural Science Foundation of Jiangsu Province

Clinical Trial Capacity Improvement Project

National Institute of Neurological Disorders and Stroke

National Science Foundation of China

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

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