PRKAA/AMPKα phosphorylation switches the role of RASAL2 from a suppressor to an activator of autophagy

Author:

Bao Yong12,Qian Christopher3,Liu Meng-Yue1,Jiang Fei1,Jiang Xiaoxiao2,Liu Huijuan2,Zhang Zhuqing2,Sun Fanghui2,Fu Ningwei4,Hou Zhaoyuan5,Ke Ya3,Li Yan5,Qian Zhong-Ming1ORCID

Affiliation:

1. Institute of Translational and Precision Medicine, Nantong University, Nantong, China

2. Department of Pharmacology and Biochemistry, Fudan University School of Pharmacy, Shanghai, China

3. School of Biomedical Sciences and Gerald Choa Neuroscience Centre, The Chinese University of Hong Kong, Shatin, Hong Kong

4. Department of Anatomy and Physiology, Shanghai Jiaotong University School of Medicine, Shanghai, China

5. Shanghai Key Laboratory for Tumor Microenvironment and Inflammation, Department of Biochemistry & Molecular Cellular Biology, Shanghai Jiaotong University School of Medicine, Shanghai, China

Funder

National Natural Science Foundation of China

Hong Kong Research Grants Council (HKRGC)—General Research Fund

Hong Kong Health Medical Research Fund

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

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