PARK2-dependent mitophagy induced by acidic postconditioning protects against focal cerebral ischemia and extends the reperfusion window-Reference-Cited by-同舟云学术

PARK2-dependent mitophagy induced by acidic postconditioning protects against focal cerebral ischemia and extends the reperfusion window

Published:2017-02-17 Issue:3 Volume:13 Page:473-485
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Shen Zhe¹,Zheng Yanrong¹,Wu Jiaying¹,Chen Ying¹,Wu Xiaoli¹,Zhou Yiting¹,Yuan Yang¹,Lu Shousheng¹,Jiang Lei¹,Qin Zhenghong²,Chen Zhong¹,Hu Weiwei¹,Zhang Xiangnan¹

Affiliation:

1. Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Department of Pharmacology, Key Laboratory of Medical Neurobiology of The Ministry of Health of China, Zhejiang Province Key Laboratory of Neurobiology, Zhejiang University, Hangzhou, China

2. Department of Pharmacology and Laboratory of Aging and Nervous Diseases, Jiangsu Key Laboratory of Translational Research and Therapy for Neuro-Psycho-Diseases, School of Pharmaceutical Science, Soochow University, Suzhou, China

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2016.1274596

Reference49 articles.

1. Brain Protection: Maybe Yes, Maybe No

2. Expansion of the Time Window for Treatment of Acute Ischemic Stroke With Intravenous Tissue Plasminogen Activator

3. 15d-Prostaglandin J2 Activates Peroxisome Proliferator-Activated Receptor-γ, Promotes Expression of Catalase, and Reduces Inflammation, Behavioral Dysfunction, and Neuronal Loss after Intracerebral Hemorrhage in Rats

4. Delayed Hypoxic Postconditioning Protects Against Cerebral Ischemia in the Mouse

5. Transient Lack of Glucose but not O2is Involved in Ischemic Postconditioning-Induced Neuroprotection

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