Autophagic reliance promotes metabolic reprogramming in oncogenic KRAS-driven tumorigenesis-Reference-Cited by-全球学者库

Autophagic reliance promotes metabolic reprogramming in oncogenic KRAS-driven tumorigenesis

Published:2018-08-21 Issue:9 Volume:14 Page:1481-1498
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Lin H. Helen¹,Chung Yiyin¹,Cheng Chun-Ting¹,Ouyang Ching²,Fu Yong¹,Kuo Ching-Ying¹,Chi Kevin K.¹,Sadeghi Maryam¹,Chu Peiguo³,Kung Hsing-Jien⁴,Li Chien-Feng⁵,Limesand Kirsten H.⁶,Ann David K.¹⁷

Affiliation:

1. Department of Diabetes and Metabolic Diseases Research

2. Center for Informatics

3. Shared Pathology Core

4. Department of Biochemistry and Molecular Medicine, UC Davis Comprehensive Cancer Center , Sacramento, CA USA

5. Department of Pathology, Chi-Mei Medical Center , Tainan, Taiwan

6. Department of Nutritional Sciences, University of Arizona , Tucson, AZ USA

7. Irell & Manella Graduate School of Biological Sciences, Beckman Research Institute, City of Hope , Duarte, CA USA

Funder

HHS | NIH | National Institute of Dental and Craniofacial Research

HHS | National Institutes of Health

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2018.1450708

Reference58 articles.

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3. Autophagy Is Critical for Pancreatic Tumor Growth and Progression in Tumors with p53 Alterations

4. Suppression of autophagy by FIP200 deletion inhibits mammary tumorigenesis

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