AMPA Receptors Endocytosis Inhibition Attenuates Cognition Deficit Via c-Fos/BDNF Signaling in Amyloid <i>β</i> Neurotoxicity-Reference-Cited by-同舟云学术

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AMPA Receptors Endocytosis Inhibition Attenuates Cognition Deficit Via c-Fos/BDNF Signaling in Amyloid β Neurotoxicity

Published:2024-07-30 Issue: Volume: Page:1-13
ISSN:0361-073X
Container-title:Experimental Aging Research
language:en
Short-container-title:Experimental Aging Research

Author:

Eyvani Kimia¹²³,Letafatkar Negin¹²³,Babaei Parvin¹²³^ORCID

Affiliation:

1. Neuroscience Research Center, Trauma Institute, Guilan University of Medical Sciences, Rasht, Iran

2. Cellular & Molecular Research Center, School of Medicine, Guilan University of Medical Sciences, Rasht, Iran

3. Department of Physiology, School of Medicine, Guilan University of Medical Sciences, Rasht, Iran

Funder

This work was supported by grant from the Research Council

Publisher

Informa UK Limited

Link

https://www.tandfonline.com/doi/pdf/10.1080/0361073X.2024.2377440

Reference55 articles.

1. Tyrosine phosphorylation of GluR2 is required for insulin-stimulated AMPA receptor endocytosis and LTD

2. The Neurotrophins and Their Role in Alzheimers Disease

3. Regulation of AMPA receptor trafficking and synaptic plasticity

4. Recent Advances on the Role of Brain-Derived Neurotrophic Factor (BDNF) in Neurodegenerative Diseases

5. NMDA and AMPA receptors dysregulation in Alzheimer's disease

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