Triptolide inhibits Th17 differentiation via controlling PKM2-mediated glycolysis in rheumatoid arthritis
Author:
Affiliation:
1. Jiangsu Key Laboratory for Pharmacology and Safety Evaluation of Chinese Materia Medica, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, Jiangsu Province, China
Funder
National Natural Science Foundation of China
the Postgraduate Research & Practice Innovation Program of Jiangsu Province
Publisher
Informa UK Limited
Subject
Pharmacology,Toxicology,General Medicine,Immunology,Immunology and Allergy
Link
https://www.tandfonline.com/doi/pdf/10.1080/08923973.2022.2086139
Reference43 articles.
1. The Pathogenesis of Rheumatoid Arthritis
2. Targeting Th17 cells in autoimmune diseases
3. Synovial fluid-derived T helper 17 cells correlate with inflammatory activity in arthritis, irrespectively of diagnosis
4. Rheumatoid arthritis bone marrow environment supports Th17 response
5. Th17 cell pathogenicity and plasticity in rheumatoid arthritis
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1. Targeting glycolytic pathway in fibroblast-like synoviocytes for rheumatoid arthritis therapy: challenges and opportunities;Inflammation Research;2023-11-08
2. Triptolide mitigates rheumatoid arthritis through Notch1/Jagged1 pathway;TROP J PHARM RES;2023
3. The potential role of T-cell metabolism-related molecules in chronic neuropathic pain after nerve injury: a narrative review;Frontiers in Immunology;2023-05-17
4. The bone-protective mechanisms of active components from TCM drugs in rheumatoid arthritis treatment;Frontiers in Pharmacology;2022-10-25
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