CXCL4 promoted the production of CD4+CD25+FOXP3+treg cells in mouse sepsis model through regulating STAT5/FOXP3 pathway
Author:
Affiliation:
1. Department of Anesthesiology, Changhai Hospital, Naval Medical University, Shanghai, PR China;
2. Intensive Care Unit, Shanghai Jiahui International Hospital, Shanghai, PR China
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
https://www.tandfonline.com/doi/pdf/10.1080/08916934.2020.1777283
Reference43 articles.
1. Immunosuppression after Sepsis: Systemic Inflammation and Sepsis Induce a Loss of Naïve T-Cells but No Enduring Cell-Autonomous Defects in T-Cell Function
2. New Approaches to Sepsis: Molecular Diagnostics and Biomarkers
3. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)
4. Neutrophil dysregulation during sepsis: an overview and update
5. High Interleukin-10 Expression in Type 2 T Helper Cells in Septic Patients
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