Frequent loss of BTG1 activity and impaired interactions with the Caf1 subunit of the Ccr4–Not deadenylase in non-Hodgkin lymphoma-Reference-Cited by-同舟云学术

Frequent loss of BTG1 activity and impaired interactions with the Caf1 subunit of the Ccr4–Not deadenylase in non-Hodgkin lymphoma

Published:2020-10-06 Issue:2 Volume:62 Page:281-290
ISSN:1042-8194
Container-title:Leukemia & Lymphoma
language:en
Short-container-title:Leukemia & Lymphoma

Author:

Almasmoum Hibah Ali¹²³^ORCID,Airhihen Blessing¹^ORCID,Seedhouse Claire²^ORCID,Winkler Gerlof Sebastiaan¹^ORCID

Affiliation:

1. School of Pharmacy, The University of Nottingham, University Park, Nottingham, UK

2. Department of Haematology, Division of Cancer and Stem Cells, School of Medicine, University of Nottingham Biodiscovery Institute, University Park, Nottingham, UK

3. Department of Laboratory Medicine, Faculty of Applied Medical Sciences, Umm Al-Qura University, Makkah, Saudi Arabia

Funder

Saudi Arabia Ministry of Higher Education

Umm Al-Qura University

Faculty for the Future Scholarship of the Schlumberger Foundation

Publisher

Informa UK Limited

Subject

Cancer Research,Oncology,Hematology

Link

https://www.tandfonline.com/doi/pdf/10.1080/10428194.2020.1827243

Reference51 articles.

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2. Tumor suppressors BTG1 and BTG2: Beyond growth control

3. Cloning of PC3B, a Novel Member of the PC3/BTG/TOB Family of Growth Inhibitory Genes, Highly Expressed in the Olfactory Epithelium

4. Tob2, a novel anti-proliferative Tob/BTG1 family member, associates with a component of the CCR4 transcriptional regulatory complex capable of binding cyclin-dependent kinases

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