Fibrillin-1 mutation contributes to Marfan syndrome by inhibiting Cav1.2-mediated cell proliferation in vascular smooth muscle cells-Reference-Cited by-同舟云学术

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Fibrillin-1 mutation contributes to Marfan syndrome by inhibiting Cav1.2-mediated cell proliferation in vascular smooth muscle cells

Published:2023-03-27 Issue:1 Volume:17 Page:
ISSN:1933-6950
Container-title:Channels
language:en
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Author:

Lin Wenfeng¹^ORCID,Xiong Jiaqi¹,Jiang Yefan¹,Liu Hao¹,Bian Jinhui¹,Wang Juejin²,Shao Yongfeng¹,Ni Buqing¹

Affiliation:

1. Department of Cardiovascular Surgery, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

2. Key Laboratory of Cardiovascular Disease and Molecular Intervention, Department of Physiology, Nanjing Medical University, Nanjing, Jiangsu, China

Funder

Jiangsu Province Hospital (the First Affiliated Hospital with Nanjing Medical University) Clinical Capacity Enhancement Project

Publisher

Informa UK Limited

Subject

Biochemistry,Biophysics

Link

https://www.tandfonline.com/doi/pdf/10.1080/19336950.2023.2192377

Reference35 articles.

1. Marfan syndrome: an update of genetics, medical and surgical management

2. Thoracic Aortic Aneurysm

3. A FBN1 3′UTR mutation variant is associated with endoplasmic reticulum stress in aortic aneurysm in Marfan syndrome

4. Vascular Smooth Muscle Cell Phenotypic Changes in Patients With Marfan Syndrome

5. Dysregulation of TGF-β activation contributes to pathogenesis in Marfan syndrome

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