The SWI/SNF ATPase BRG1 stimulates DNA end resection and homologous recombination by reducing nucleosome density at DNA double strand breaks and by promoting the recruitment of the CtIP nuclease-Reference-Cited by-同舟云学术

The SWI/SNF ATPase BRG1 stimulates DNA end resection and homologous recombination by reducing nucleosome density at DNA double strand breaks and by promoting the recruitment of the CtIP nuclease

Published:2020-10-12 Issue:22 Volume:19 Page:3096-3114
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Hays Emily¹²,Nettleton Elizabeth³,Carter Caitlin³,Morales Mariangel²⁴,Vo Lynn³,Passo Max²⁴,Vélez-Cruz Renier¹²⁵^ORCID

Affiliation:

1. Department of Biochemistry and Molecular Genetics, Midwestern University, Downers Grove, IL, USA

2. College of Graduate Studies, Midwestern University, Downers Grove, IL, USA

3. Chicago College of Osteopathic Medicine, Midwestern University, Downers Grove, IL, USA

4. Biomedical Sciences Program, Midwestern University, Downers Grove, IL, USA

5. Chicago College of Optometry, Midwestern University, Downers Grove, IL, USA

Funder

Midwestern University

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2020.1831256

Reference63 articles.

1. Homologous Recombination and the Formation of Complex Genomic Rearrangements

2. Main steps in DNA double-strand break repair: an introduction to homologous recombination and related processes

3. Drugging the Cancers Addicted to DNA Repair

4. Therapeutic targeting and patient selection for cancers with homologous recombination defects

5. Exploiting DNA repair defects for novel cancer therapies

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