Spy1 induces de-ubiquitinating of RIP1 arrest and confers glioblastoma's resistance to tumor necrosis factor (TNF-α)-induced apoptosis through suppressing the association of CLIPR-59 and CYLD

Author:

Ding Zongmei12,Liu Yonghua1,Yao Li3,Wang Donglin4,Zhang Jianguo4,Cui Gang5,Yang Xiaojing1,Huang Xianting1,Liu Fang167,Shen Aiguo167

Affiliation:

1. Jiangsu Province Key Laboratory for Inflammation and Molecular Drug Target; Nantong University; Nantong, Jiangsu, PR China

2. Department of Pathology; Subei People's Hospital; Yangzhou, Jiangsu, PR China

3. Department of Immunology; Wuxi Medical College; Jiangnan University; Wuxi, Jiangsu, PR China

4. Department of Pathology; Medical College; Nantong University; Nantong, Jiangsu, PR China

5. Department of Neurosurgery; The First Affiliated Hospital of Soochow University; Suzhou, Jiangsu, PR China

6. Jiangsu Province Key Laboratory of Neuroregeneration; Nantong University; Nantong, Jiangsu, PR China

7. Co-innovation Center of Neuroregeneration; Nantong University; Nantong, Jiangsu, PR China

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

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