Deciphering the mechanism of HM43239 inhibiting the mutant F691L resistant to gilteritinib in FMS-like tyrosine kinase 3-Reference-Cited by-同舟云学术

Deciphering the mechanism of HM43239 inhibiting the mutant F691L resistant to gilteritinib in FMS-like tyrosine kinase 3

Published:2023-06-29 Issue: Volume: Page:1-10
ISSN:0739-1102
Container-title:Journal of Biomolecular Structure and Dynamics
language:en
Short-container-title:Journal of Biomolecular Structure and Dynamics

Author:

Wang Zhiwei¹^ORCID,An Yu²,Wang Jian³,Lu Jinghua¹

Affiliation:

1. College of Pharmacy, Jinzhou Medical University, Linghe District, Jinzhou, China

2. Department of Open Education, Jinzhou Open University, Linghe District, Jinzhou, China

3. Key Laboratory of Structure-Based Drug Design and Discovery, Ministry of Education, Shenyang Pharmaceutical University, Shenhe District, Shenyang, China

Funder

the Director’s Fund Projects from Liaoning Provincial Key Laboratory of Marine Bioactive Substances and Technological Innovation Center of Liaoning Pharmaceutical Action and Quality Evaluation

Publisher

Informa UK Limited

Subject

Molecular Biology,General Medicine,Structural Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/07391102.2023.2229447

Reference31 articles.

1. Identification of novel FLT-3 Asp835 mutations in adult acute myeloid leukaemia

2. Abstract 1257: HM43239, a novel FLT3 inhibitor, has the potential to inhibit mutations resistant to FLT3 inhibitors

3. Identification of a novel type of ITD mutations located in nonjuxtamembrane domains of the FLT3 tyrosine kinase receptor

4. Orally administered colony stimulating factor 1 receptor inhibitor PLX3397 in recurrent glioblastoma: an Ivy Foundation Early Phase Clinical Trials Consortium phase II study

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