Hyperoside ameliorates cisplatin-induced acute kidney injury by regulating the expression and function of Oat1-Reference-Cited by-同舟云学术

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Hyperoside ameliorates cisplatin-induced acute kidney injury by regulating the expression and function of Oat1

Published:2023-09-02 Issue:8-9 Volume:53 Page:559-571
ISSN:0049-8254
Container-title:Xenobiotica
language:en
Short-container-title:Xenobiotica

Author:

Yuan Wenjing¹,Kou Shanshan¹,Ma Ying²,Qian Yusi¹,Li Xinyu¹,Chai Yuanyuan¹,Jiang Zhenzhou¹,Zhang Luyong³,Sun Lixin¹,Huang Xin¹^ORCID

Affiliation:

1. Key Laboratory of Drug Quality Control and Pharmacovigilance, Ministry of Education, China Pharmaceutical University, Nanjing, PR China

2. Foreign Language Teaching Department, Nanjing University of Chinese Medicine, Nanjing, PR China

3. Center for Drug Research and Development, Guangdong Pharmaceutical University, Guangzhou, PR China

Funder

‘Double First-Class’ University Projects

The Open Project Program of the MOE Key Laboratory of Drug Quality Control and Pharmacovigilance

Publisher

Informa UK Limited

Subject

Health, Toxicology and Mutagenesis,Pharmacology,Toxicology,Biochemistry,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/00498254.2023.2270046

Reference56 articles.

1. Hyperoside pre-treatment prevents glomerular basement membrane damage in diabetic nephropathy by inhibiting podocyte heparanase expression

2. Role of Resveratrol on Indoxyl Sulfate-Induced Endothelial Hyperpermeability via Aryl Hydrocarbon Receptor (AHR)/Src-Dependent Pathway

3. Gut-Microbiota-Metabolite Axis in Early Renal Function Decline

4. Gut-derived uremic toxin handling in vivo requires OAT-mediated tubular secretion in chronic kidney disease

5. Hyperoside alleviates adriamycin-induced podocyte injury via inhibiting mitochondrial fission

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