Hepatitis B virus mutation pattern rtL180M+A181C+M204V may contribute to entecavir resistance in clinical practice-Reference-Cited by-全球学者库

Hepatitis B virus mutation pattern rtL180M+A181C+M204V may contribute to entecavir resistance in clinical practice

Published:2019-01-01 Issue:1 Volume:8 Page:354-365
ISSN:2222-1751
Container-title:Emerging Microbes & Infections
language:en
Short-container-title:Emerging Microbes & Infections

Author:

Liu Yan¹,Zhou Yi²,Li Xiaodong¹,Niu Ming³,Chen Rongjuan¹,Shao Jinman¹,Si Lanlan¹,Luo Dan²,Lin Yayun²,Li Le¹,Zhang Kai²,Xiao Xiaohe³,Xu Zhihui¹,Liu Min²,Lu Mengji⁴,Zoulim Fabien⁵⁶,Xu Dongping¹

Affiliation:

1. Institute of Infectious Diseases, Beijing 302 Hospital, Beijing, People’s Republic of China

2. Department of Infectious Diseases, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an, People’s Republic of China

3. Institute of Chinese Medicine, Beijing 302 Hospital, Beijing, People’s Republic of China

4. Institute of Virology, University Hospital of Essen, University of Duisburg-Essen, Essen, Germany

5. Univ Lyon, Universite Claude Bernard Lyon 1, INSERM 1052, CNRS 5286, Centre Leon Berard, Centre de recherche en cancerologie de Lyon, Lyon, France

6. Department of Hepatology, Groupement Hospitalier Nord, Hospices Civils de Lyon, Lyon, France

Funder

National Natural Science Foundation of China

Publisher

Informa UK Limited

Subject

Virology,Infectious Diseases,Drug Discovery,General Medicine,Immunology,Microbiology,Parasitology,Epidemiology

Link

https://www.tandfonline.com/doi/pdf/10.1080/22221751.2019.1584018

Reference39 articles.

1. EASL 2017 Clinical Practice Guidelines on the management of hepatitis B virus infection

2. The global burden of liver disease: The major impact of China

3. Reasons to consider earlier treatment of chronic HBV infections

4. Antiviral drug-resistant HBV: Standardization of nomenclature and assays and recommendations for management

5. Hepatitis B virus quasispecies susceptibility to entecavir confirms the relationship between genotypic resistance and patient virologic response

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