Author:
Tran Kiet Anh,Li Jianping,Khon Huynh Chan,Takei Masahiro
Reference11 articles.
1. Jacob, H.S., Ingbar, S.H., Jandl, J.H.: Oxidative hemolysis and erythrocyte metabolism in hereditary acatalasia. J. Clin. Inv. 44(7), 1187 (1965)
2. Reiter, C.D., et al.: Cell-free hemoglobin limits nitric oxide bioavailability in sickle-cell disease. Nat. Med. 8(12), 1383 (2002), December
3. Emerson, C.P., et al.: Studies on the destruction of red blood cells: IX. Quantitative methods for determining the osmotic and mechanical fragility of red cells in the peripheral blood and splenic pulp; the mechanism of increased hemolysis in hereditary spherocytosis (Congenital Hemolytic Jaundice) as related to the functions of the spleen. AMA Arch. Intern. Med. 97(1) (1956), 1-38 LNCS Homepage, http://www.springer.com/lncs . Last Accessed 2016/11/21
4. Clark, I.A., Hunt, N.H.: Evidence for reactive oxygen intermediates causing hemolysis and parasite death in Malaria. Infect. Immun. 39(1), 1–6 (1983)
5. Linden, J.V., et al.: Transfusion errors in New York State: an analysis of 10 years’ experience. Transfusion 40(10), 1207–1213 (2000)