Ibrutinib Delays ALS Installation and Increases Survival of SOD1G93A Mice by Modulating PI3K/mTOR/Akt Signaling
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology,Immunology and Allergy,Neuroscience (miscellaneous)
Link
https://link.springer.com/content/pdf/10.1007/s11481-023-10068-9.pdf
Reference59 articles.
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2. Barrientos J, Rai K (2013) Ibrutinib: a novel Bruton’s tyrosine kinase inhibitor with outstanding responses in patients with chronic lymphocytic leukemia. Leuk Lymphoma 54(8):1817–1820. https://doi.org/10.3109/10428194.2013.796049
3. Bendotti C, Carrì MT (2004) Lessons from models of SOD1-linked familial ALS. Trends Mol Med 10(8):393–400. https://doi.org/10.1016/j.molmed.2004.06.009
4. Calvo A, Moglia C, Balma M, Chio A (2010) Involvement of immune response in the pathogenesis of amyotrophic lateral sclerosis: a therapeutic opportunity? CNS & neurological Disorders-Drug targets (formerly current drug Targets-CNS &. Neurol Disorders) 9(3):325–330
5. Comley LH, Nijssen J, Frost-Nylen J, Hedlund E (2016) Cross-disease comparison of amyotrophic lateral sclerosis and spinal muscular atrophy reveals conservation of selective vulnerability but differential neuromuscular junction pathology. J Comp Neurol 524(7):1424–1442. https://doi.org/10.1002/cne.23917
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