Dysregulation of long non-coding RNAs in Takayasu arteritis: A proof-of-concept study
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine,Rheumatology
Link
https://link.springer.com/content/pdf/10.1007/s10067-024-06880-0.pdf
Reference23 articles.
1. Castillo-Martínez D, Amezcua-Guerra LM (2012) Self-reactivity against stress-induced cell molecules: The missing link between Takayasu’s arteritis and tuberculosis? Med Hypotheses 78:485–488. https://doi.org/10.1016/j.mehy.2012.01.012
2. Joseph G, Goel R, Thomson VS, Joseph E, Danda D (2022) Takayasu arteritis: JACC focus seminar 3/4. J Am Coll Cardiol 81(2):172–186. https://doi.org/10.1016/j.jacc.2022.09.051
3. Wen D, Feng L, Du X, Dong JZ, Ma CS (2023) Biomarkers in Takayasu arteritis. Int J Cardiol 371:413–417. https://doi.org/10.1016/j.ijcard.2022.08.058
4. Amezcua-Guerra LM, Sánchez-Muñoz F, Pichardo-Ontiveros E, González-Ramírez J, Martínez-Martínez LA, Juárez-Vicuña Y (2022) Interferon-alpha regulates expression of lncRNA MALAT1 and interferon-stimulated genes, as well as chemokine production, in primary Sjögren's syndrome. Clin Exp Rheumatol 40:2275–2282. https://doi.org/10.55563/clinexprheumatol/ggkc9t
5. Yang K, Tang J, Li H, Zhang H, Ding J, Li Z (2023) Luo J (2023) LncRNAs in Kawasaki disease and Henoch-Schönlein purpura: Mechanisms and clinical applications. Mol Cell Biochem. https://doi.org/10.1007/s11010-023-04832-x
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