Role of Oxygen Free Radicals, Nitric Oxide and Mitochondria in Mediating Cardiac Alterations During Liver Cirrhosis Induced by Thioacetamide

Author:

Amirtharaj G. Jayakumar,Natarajan Sathish Kumar,Pulimood Anna,Balasubramanian K. A.,Venkatraman Aparna,Ramachandran Anup

Funder

Department of Biotechnology, Ministry of Science and Technology

Council of Scientific and Industrial Research

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology

Reference45 articles.

1. Wang, T., Shankar, K., Ronis, M. J., & Mehendale, H. M. (2000). Potentiation of thioacetamide liver injury in diabetic rats is due to induced CYP2E1. Journal of Pharmacology and Experimental Therapeutics, 294, 473–479.

2. Ambrose, A. M., De, E. F., & Rather, L. J. (1949). Toxicity of thioacetamide in rats. The Journal of Industrial Hygiene and Toxicology, 31, 158–161.

3. Fitzhugh, O. G., & Nelson, A. A. (1948). Liver tumors in rats fed thiourea or thioacetamide. Science, 108, 626–628.

4. Rather, L. J. (1951). Experimental alteration of nuclear and cytoplasmic components of the liver cell with thioacetamide. I. Early onset and reversibility of volume changes of the nucleolus, nucleus and cytoplasm. Bulletin of the Johns Hopkins Hospital, 88, 38–58.

5. Al-Hamoudi, W. K. (2010). Cardiovascular changes in cirrhosis: Pathogenesis and clinical implications. Saudi Journal of Gastroenterology, 16, 145–153.

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