TGF-β1 Neutralization Improves Pregnancy Outcomes by Restoring Endometrial Receptivity in Mice with Adenomyosis
Author:
Funder
MOST research fund
E-Da Hospital Research Fund
Publisher
Springer Science and Business Media LLC
Subject
Obstetrics and Gynaecology
Link
https://link.springer.com/content/pdf/10.1007/s43032-020-00308-1.pdf
Reference60 articles.
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2. Leyendecker G, Wildt L, Mall G. The pathophysiology of endometriosis and adenomyosis: tissue injury and repair. Arch Gynecol Obstet. 2009;280(4):529–38. https://doi.org/10.1007/s00404-009-1191-0.
3. Templeman C, Marshall SF, Ursin G, Horn-Ross PL, Clarke CA, Allen M, et al. Adenomyosis and endometriosis in the California teachers study. Fertil Steril. 2008;90(2):415–24. https://doi.org/10.1016/j.fertnstert.2007.06.027.
4. Bergeron C, Amant F, Ferenczy A. Pathology and physiopathology of adenomyosis. Best Pract Res Clin Obstet Gynaecol. 2006;20(4):511–21. https://doi.org/10.1016/j.bpobgyn.2006.01.016.
5. Shen M, Liu X, Zhang H, Guo SW. Transforming growth factor beta1 signaling coincides with epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis in mice. Hum Reprod. 2016;31(2):355–69. https://doi.org/10.1093/humrep/dev314.
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