IL-12/23-targeting in seborrheic dermatitis patients leads to long-lasting response
Author:
Publisher
Springer Science and Business Media LLC
Subject
Dermatology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00403-023-02680-9.pdf
Reference5 articles.
1. Adalsteinsson JA, Kaushik S, Muzumdar S, Guttman-Yassky E, Ungar J (2020) An update on the microbiology, immunology and genetics of seborrheic dermatitis. Exp Dermatol 29(5):481–489. https://doi.org/10.1111/exd.14091
2. Faergemann J, Bergbrant IM, Dohse M, Scott A, Westgate G (2001) Seborrhoeic dermatitis and Pityrosporum (Malassezia) folliculitis: characterization of inflammatory cells and mediators in the skin by immunohistochemistry. Br J Dermatol 144(3):549–556. https://doi.org/10.1046/j.1365-2133.2001.04082.x
3. Molinero LL, Gruber M, Leoni J, Woscoff A, Zwirner NW (2003) Up-regulated expression of MICA and proinflammatory cytokines in skin biopsies from patients with seborrhoeic dermatitis. Clin Immunol 106(1):50–54. https://doi.org/10.1016/s1521-6616(03)00003-2
4. Sparber F, De Gregorio C, Steckholzer S et al (2019) The skin commensal yeast malassezia triggers a type 17 response that coordinates anti-fungal immunity and exacerbates skin inflammation. Cell Host Microb 25(3):389-403 e6. https://doi.org/10.1016/j.chom.2019.02.002
5. Griffiths CEM, Armstrong AW, Gudjonsson JE, Barker J (2021) Psoriasis Lancet 397(10281):1301–1315. https://doi.org/10.1016/S0140-6736(20)32549-6
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