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Inflammation and fibrosis in murine models of heart failure

Published:2019-03-18 Issue:3 Volume:114 Page:
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Bacmeister Lucas,Schwarzl Michael,Warnke Svenja,Stoffers Bastian,Blankenberg Stefan,Westermann Dirk,Lindner Diana

Funder

Deutsches Zentrum für Herz-Kreislaufforschung

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00395-019-0722-5.pdf

Reference294 articles.

1. Aartsen WM, Pelsers MM, Hermens WT, Glatz JF, Daemen MJ, Smits JF (2000) Heart fatty acid binding protein and cardiac troponin T plasma concentrations as markers for myocardial infarction after coronary artery ligation in mice. Pflugers Arch 439:416–422. https://doi.org/10.1007/s004249900180

2. Adiarto S, Heiden S, Vignon-Zellweger N, Nakayama K, Yagi K, Yanagisawa M, Emoto N (2012) ET-1 from endothelial cells is required for complete angiotensin II-induced cardiac fibrosis and hypertrophy. Life Sci 91:651–657. https://doi.org/10.1016/j.lfs.2012.02.006

3. Alard JE, Ortega-Gomez A, Wichapong K, Bongiovanni D, Horckmans M, Megens RT, Leoni G, Ferraro B, Rossaint J, Paulin N, Ng J, Ippel H, Suylen D, Hinkel R, Blanchet X, Gaillard F, D’Amico M, von Hundelshausen P, Zarbock A, Scheiermann C, Hackeng TM, Steffens S, Kupatt C, Nicolaes GA, Weber C, Soehnlein O (2015) Recruitment of classical monocytes can be inhibited by disturbing heteromers of neutrophil HNP1 and platelet CCL5. Sci Transl Med 7:317ra196. https://doi.org/10.1126/scitranslmed.aad5330

4. Alex L, Frangogiannis NG (2018) The cellular origin of activated fibroblasts in the infarcted and remodeling myocardium. Circ Res 122:540–542. https://doi.org/10.1161/CIRCRESAHA.118.312654

5. Andersson L, Scharin Tang M, Lundqvist A, Lindbom M, Mardani I, Fogelstrand P, Shahrouki P, Redfors B, Omerovic E, Levin M, Boren J, Levin MC (2015) Rip2 modifies VEGF-induced signalling and vascular permeability in myocardial ischaemia. Cardiovasc Res 107:478–486. https://doi.org/10.1093/cvr/cvv186

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