Increased double-strand breaks in aged mouse male germ cells may result from changed expression of the genes essential for homologous recombination or nonhomologous end joining repair-Reference-Cited by-同舟云学术

Increased double-strand breaks in aged mouse male germ cells may result from changed expression of the genes essential for homologous recombination or nonhomologous end joining repair

Published:2022-10-15 Issue:2 Volume:159 Page:127-147
ISSN:0948-6143
Container-title:Histochemistry and Cell Biology
language:en
Short-container-title:Histochem Cell Biol

Author:

Talibova Gunel,Bilmez Yesim,Ozturk Saffet

Funder

The Scientific and Technological Research Council of Turkey

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Medical Laboratory Technology,Molecular Biology,Histology

Link

https://link.springer.com/content/pdf/10.1007/s00418-022-02157-2.pdf

Reference98 articles.

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2. Ahmed EA, van der Vaart A, Barten A, Kal HB, Chen J, Lou Z, Minter-Dykhouse K, Bartkova J, Bartek J, de Boer P, de Rooij DG (2007) Differences in DNA double strand breaks repair in male germ cell types: lessons learned from a differential expression of Mdc1 and 53BP1. DNA Repair (Amst) 6(9):1243–1254. https://doi.org/10.1016/j.dnarep.2007.02.011

3. Ahnesorg P, Smith P, Jackson SP (2006) XLF interacts with the XRCC4-DNA ligase IV complex to promote DNA nonhomologous end-joining. Cell 124(2):301–313. https://doi.org/10.1016/j.cell.2005.12.031

4. Anderson RA Jr, Phillips JF, Berryman SH, Zaneveld LJ (1989) Testosterone production by the prepubertal mouse testis is not depressed by ethanol. Reprod Toxicol 3(2):91–100

5. Baumann P, West SC (1998) Role of the human RAD51 protein in homologous recombination and double-stranded-break repair. Trends Biochem Sci 23(7):247–251. https://doi.org/10.1016/s0968-0004(98)01232-8

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