Vulnerability to a Metabolic Challenge Following Perinatal Asphyxia Evaluated by Organotypic Cultures: Neonatal Nicotinamide Treatment-Reference-Cited by-同舟云学术

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Vulnerability to a Metabolic Challenge Following Perinatal Asphyxia Evaluated by Organotypic Cultures: Neonatal Nicotinamide Treatment

Published:2017-06-19 Issue:3 Volume:32 Page:426-443
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Perez-Lobos R.,Lespay-Rebolledo C.,Tapia-Bustos A.,Palacios E.,Vío V.,Bustamante D.,Morales P.,Herrera-Marschitz M.

Funder

FONDECYT-Chile

Millenium Institute-Chile

CONICYT-Chile

MECESUP-Chile

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

http://link.springer.com/article/10.1007/s12640-017-9755-4/fulltext.html

Reference98 articles.

1. Allende-Castro C, Espina-Marchant P, Bustamante D, Rojas-Mancilla E, Neira T, Gutierrez-Hernandez MA, Esmar D, Valdes JL, Morales P, Gebicke-Haerter PJ, Herrera-Marschitz M (2012) Further studies on the hypothesis of PARP-1 inhibition as strategy for lessening the long-term effects produced by perinatal asphyxia: effects of nicotinamide and theophylline on PARP-1 activity in brain and peripheral tissue. Neurotox Res 22:79–90

2. Amoroso S, Tortiglione A, Secondo A, Catalano A, Montagni S, Di Renzo G, Annunziato L (2000) Sodium nitroprusside prevents chemical hypoxia-induced cell death through iron ions stimulating the activity of the Na+-Ca2+ exchanger in C6 glioma cells. J Neurochem 74:1505–1513

3. Ara J, Fekete S, Frank M, Golden JA, Pleasure D, Valencia I (2011) Hypoxic-preconditioning induces neuroprotection against hypoxia-ischemia in newborn piglet brain. Neurobiol Dis 43:473–485

4. Aschbacher K, O’Donovan A, Wolkowitz OM, Dhabhar FS, Su Y, Epel E (2013) Good stress, bad stress and oxidative stress: insights from anticipatory cortisol reactivity. Psychoneuroendocrinology 38:1698–1708

5. Bai J-Z, Lipski J (2010) Diffrential expression of TRPM2 and TRPV4 channels and their potential role in oxidative stress-induced cell death in organotypic hippocampal culture. Neurotoxicology 31:204–214

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