Novel Interactive Partners of Neuroligin 3: New Aspects for Pathogenesis of Autism-Reference-Cited by-同舟云学术

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Novel Interactive Partners of Neuroligin 3: New Aspects for Pathogenesis of Autism

Published:2014-12-03 Issue:1 Volume:56 Page:89-101
ISSN:0895-8696
Container-title:Journal of Molecular Neuroscience
language:en
Short-container-title:J Mol Neurosci

Author:

Shen Chen,Huo Li-rong,Zhao Xin-liang,Wang Pei-rong,Zhong Nanbert

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s12031-014-0470-9.pdf

Reference53 articles.

1. Aki T, Funakoshi T, Nishida-Kitayama J, Mizukami Y (2008) TPRA40/GPR175 regulates early mouse embryogenesis through functional membrane transport by Sjögren’s syndrome-associated protein NA14. J Cell Physiol 217(1):194–206

2. Auerbach BD, Osterweil EK, Bear MF (2011) Mutations causing syndromic autism define an axis of synaptic pathophysiology. Nature 480(7375):63–68

3. Avdjieva-Tzavella D, Mihailova S, Lukanov C, Naumova E, Simeonov E, Tincheva R, Toncheva D (2012) Mitochondrial DNA mutations in two Bulgarian children with autistic spectrum disorders. Balk J Med Genet 15(2):47–54

4. Baudouin SJ, Gaudias J, Gerharz S, Hatstatt L, Zhou K, Punnakkal P, Tanaka KF, Spooren W, Hen R, De Zeeuw CI, Vogt K, Scheiffele P (2012) Shared synaptic pathophysiology in syndromic and nonsyndromic rodent models of autism. Science 338(6103):128–132

5. Bemben MA, Shipman SL, Hirai T, Herring BE, Li Y, Badger JD, Nicoll RA, Diamond JS, Roche KW (2014) ,CaMKII phosphorylation of neuroligin-1 regulates excitatory synapses. Nat Neurosci 17(1):56–64

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