Inhibition of Nucleostemin Upregulates CDX2 Expression in HT29 Cells in Response to Bile Acid Exposure: Implications in the Pathogenesis of Barrett’s Esophagus
Author:
Publisher
Springer Science and Business Media LLC
Subject
Gastroenterology,Surgery
Link
http://link.springer.com/content/pdf/10.1007/s11605-009-0899-2.pdf
Reference50 articles.
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3. Paulson TG, Reid BJ. Focus on Barrett’s esophagus and esophageal adenocarcinoma. Cancer Cell 2004;6:1–16. doi: 10.1016/j.ccr.2004.06.021 .
4. Lagergren J, Bergstrom R, Lindgren A, Nyrén O. Symptomatic gastroesophageal reflux as a risk factor for esophageal adenocarcinoma. N Engl J Med 1999;340:825–831. doi: 10.1056/NEJM199903183401101 .
5. Tsai RY, McKay RD. A nucleolar mechanism controlling cell proliferation in stem cells and cancer cells. Genes Dev 2002;16:2991–3003. doi: 10.1101/gad.55671 .
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Knockdown of Nucleostemin can inhibit the Proliferation of Esophageal Carcinoma Cells in Vitro Through Upregulating P21;HEPATO-GASTROENTEROL;2014
2. In vitro Study of Nucleostemin Gene as a Potential Therapeutic Target for Human Lung Carcinoma;BIOMED ENVIRON SCI;2012
3. Systematic review: the role of bile acids in the pathogenesis of gastro-oesophageal reflux disease and related neoplasia;Alimentary Pharmacology & Therapeutics;2011-05-25
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