Midostaurin for FLT3-mutated AML: a real-world analysis of effectiveness and infection risk at a single center
Author:
Publisher
Springer Science and Business Media LLC
Subject
Hematology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00277-024-05614-1.pdf
Reference7 articles.
1. Kottaridis PD, Gale RE, Frew ME et al (2001) The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the United Kingdom Medical Research Council AML 10 and 12 trials. Blood 98:1752–1759. https://doi.org/10.1182/BLOOD.V98.6.1752
2. Gale RE, Green C, Allen C et al (2008) The impact of FLT3 internal tandem duplication mutant level, number, size, and interaction with NPM1 mutations in a large cohort of young adult patients with acute myeloid leukemia. Blood 111:2776–2784. https://doi.org/10.1182/BLOOD-2007-08-109090
3. Bacher U, Haferlach C, Kern W et al (2008) Prognostic relevance of FLT3-TKD mutations in AML: the combination matters—an analysis of 3082 patients. Blood 111:2527–2537. https://doi.org/10.1182/BLOOD-2007-05-091215
4. Fröhling S, Schlenk RF, Breitruck J et al (2002) Prognostic significance of activating FLT3 mutations in younger adults (16 to 60 years) with acute myeloid leukemia and normal cytogenetics: A study of the AML study group Ulm. Blood 100:4372–4380. https://doi.org/10.1182/blood-2002-05-1440
5. Stone RM, Mandrekar SJ, Sanford BL et al (2017) Midostaurin plus Chemotherapy for Acute Myeloid Leukemia with a FLT3 Mutation. N Engl J Med 377:454–464. https://doi.org/10.1056/NEJMOA1614359/SUPPL_FILE/NEJMOA1614359_DISCLOSURES.PDF
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