The podocyte as a target: cyclosporin A in the management of the nephrotic syndrome caused by WT1 mutations
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology and Child Health
Link
http://link.springer.com/content/pdf/10.1007/s00431-011-1397-6.pdf
Reference54 articles.
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2. Ariyaratana S, Loeb DM (2007) The role of the Wilms tumour gene (WT1) in normal and malignant haematopoiesis. Expert Rev Mol Med 9:1–17
3. Aucella F, Bisceglia L, De Bonis P et al (2006) WT1 mutations in nephrotic syndrome revisited. high prevalence in young girls, associations and renal phenotypes. Pediatr Nephrol 21:1393–1398
4. Baker AJ, Mooney A, Hughes J et al (1994) Mesangial cell apoptosis: the major mechanism for resolution of glomerular hypercellularity in experimental mesangial proliferative nephritis. J Clin Invest 94:2105–2116
5. Barbaux S, Niaudet P, Gubler MC et al (1997) Donor splice-site mutations in WT1 are responsible for Frasier syndrome. Nat Genet 17:467–470
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