Identification of BACH2 as a susceptibility gene for Graves’ disease in the Chinese Han population based on a three-stage genome-wide association study

Author:

Liu Wei, ,Wang Hai-Ning,Gu Zhao-Hui,Yang Shao-Ying,Ye Xiao-Ping,Pan Chun-Ming,Zhao Shuang-Xia,Xue Li-Qiong,Xie Hui-Jun,Yu Sha-Sha,Guo Cui-Cui,Du Wen-Hua,Liang Jun,Zhang Xiao-Mei,Yuan Guo-Yue,Li Chang-Gui,Su Qing,Gao Guan-Qi,Song Huai-Dong

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics

Reference36 articles.

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2. Bradfield JP, Qu HQ, Wang K, Zhang H, Sleiman PM, Kim CE, Mentch FD, Qiu H, Glessner JT, Thomas KA, Frackelton EC, Chiavacci RM, Imielinski M, Monos DS, Pandey R, Bakay M, Grant SF, Polychronakos C, Hakonarson H (2011) A genome-wide meta-analysis of six type 1 diabetes cohorts identifies multiple associated loci. PLoS Genet 7:e1002293. doi: 10.1371/journal.pgen.1002293

3. Brent GA (2010) Environmental exposures and autoimmune thyroid disease. Thyroid 20:755–761. doi: 10.1089/thy.2010.1636

4. Brix TH, Kyvik KO, Christensen K, Hegedus L (2001) Evidence for a major role of heredity in Graves’ disease: a population-based study of two Danish twin cohorts. J Clin Endocrinol Metab 86:930–934

5. Carella C, Mazziotti G, Sorvillo F, Piscopo M, Cioffi M, Pilla P, Nersita R, Iorio S, Amato G, Braverman LE, Roti E (2006) Serum thyrotropin receptor antibodies concentrations in patients with Graves’ disease before, at the end of methimazole treatment, and after drug withdrawal: evidence that the activity of thyrotropin receptor antibody and/or thyroid response modify during the observation period. Thyroid 16:295–302. doi: 10.1089/thy.2006.16.295

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